kidney stones procedures do not increase chronic kidney disease (CKD) Risk: Study ; Home “ kidney Health “
Kidney stone procedures do not pose chronic renal disease (ERC) risk. The procedure for the treatment of urolithiasis has been found to increase the risk of increased serum creatinine, but not chronic kidney disease.
The researchers conducted a retrospective cohort observational study, which included 1,340 patients diagnosed with kidney stones – urolithiasis. Of these, 446 underwent urological procedures. Mean follow-up was 9.8 years for those who underwent the procedure and 9.5 years for those who did not.
Those who underwent the procedure had a 49 percent increase in serum creatinine, but no significant difference in the risk of chronic kidney disease between the two groups.
elevated serum creatinine levels may be the result of the procedure, but more often than not a result of secondary to urolithiasis event itself renal injury.
The authors concluded that “urological procedures can protect people with urolithiasis most important events of renal dysfunction in the long term.”
Urolithiasis is the process of formation of kidney stones, bladder or urethra. Kidney stones are caused by the crystallisation of minerals, which occurs when there is insufficient urine levels or when forming salt crystals are present. Kidney stones can be caused by many different things, including calcium oxalate, uric acid, cysteine, or xanthine.
Based on the cause, there are different types of kidney stones, including uric acid stones, calcium stones (the most common kidney stones), struvite, cystine stones, and stones xanthine.
Kidney stones are described as one of the most painful medical conditions you may have. Kidney stones are caused by oxalate, which is commonly found in humans and plants. Oxalate is not necessarily required by the body, and excess thereof can result in kidney stones.
In plants, I oxalate helps eliminate excess calcium, hence its high oxalate content. For humans, oxalate can function as a “prebiotic”, meaning that contributes to the good bacteria in the gut.
When oxalate ingested with food , which is broken down by the digestive system and is released through our feces or urine. On its way through the intestines, oxalate can bind with calcium and get excreted through our feces. But when high levels of oxalate continuously pass through the kidneys, this is when kidney stones may develop. calcium oxalate kidney stones are the most common variety of kidney stones.
risk factors and symptoms Urolithiasis
The risk of urolithiasis increases with any factor leading to urinary stasis, a prolonged accumulation of urine.
Risk factors for urolithiasis include:
- The male
- Being Native American, African, or Israeli
- Family history production of excess protein
- Having medical history as bowel disease, recurrent urinary tract infections, and cystinuria
- After a poor diet and drink enough fluids
- living in tropical climates, mountain areas, or in the desert
- Being certain drugs, such as ephedrine, guaifenesin, thiazide, indinavir, and allopurinol
urolithiasis symptoms depend on the location of the stones. You may notice changes in urine, painful urination, pain in the back or lower abdomen, and even blood in the urine. If you experience any of these symptoms, you should consult a doctor immediately to minimize the risk of complications.
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